I've read the report through quickly and one of the observations was for the increase in stillbirths. The definition was changed in 2019 from baby delivered weighing over 500 gr to that plus 24 weeks gestation and authors say this would increase numbers but in comparison to 2019, 2020 showed no increase but shows ave 10% increase since. The miscarriages happening in first trimester must therefore be accounted for in the much lower live birth figures. I hope and pray that no permanent damage has been done to these women.
Is the spike protein acting as a prion with regard to hemoglobin molecules? And is porphyria being induced?
If the answers are 'yes', this is very bad news.
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Here’s a key paragraph from that article:
So the bottom line of all of this information is this: the virus infects the RBCs using spike protein via the CD147 receptor on red blood cells which causes hemolysis (rupture of the red blood cell). This causes the release of massive amounts of hemoglobin. Then the spike protein, due to its amyloidogenic peptides, triggers mis-folding of the hemoglobin into amyloid fibrils causing subsequent blood clots. The blood clots would be enhanced due to antibodies (Ag:Ab complexes).
I've read the report through quickly and one of the observations was for the increase in stillbirths. The definition was changed in 2019 from baby delivered weighing over 500 gr to that plus 24 weeks gestation and authors say this would increase numbers but in comparison to 2019, 2020 showed no increase but shows ave 10% increase since. The miscarriages happening in first trimester must therefore be accounted for in the much lower live birth figures. I hope and pray that no permanent damage has been done to these women.
Is the spike protein acting as a prion with regard to hemoglobin molecules? And is porphyria being induced?
If the answers are 'yes', this is very bad news.
--
Here’s a key paragraph from that article:
So the bottom line of all of this information is this: the virus infects the RBCs using spike protein via the CD147 receptor on red blood cells which causes hemolysis (rupture of the red blood cell). This causes the release of massive amounts of hemoglobin. Then the spike protein, due to its amyloidogenic peptides, triggers mis-folding of the hemoglobin into amyloid fibrils causing subsequent blood clots. The blood clots would be enhanced due to antibodies (Ag:Ab complexes).
https://jessicar.substack.com/p/is-the-spike-protein-acting-as-a
Excellent article. Many thanks. I hope a large number of people see this.
Thanks for the excellent article. It is very well done.